Introduction:
Cannabis is not just the most abused illicit drug in the United States (Gold, Frost Pineda, & Jacobs, 2004; NIDA, 2010) it is in fact the most abused illegal drug globally (UNODC, 2010). In the United States it is a schedule-I component meaning that it's legally deemed as having no medical use and it is highly addictive (US DEA, 2010). Doweiko (2009) explains that only some cannabis has abuse potential. He therefore suggests making use of the standard terminology marijuana when discussing cannabis with abuse potential. For the benefit of clarity this terminology is used in this paper also.
Nowadays, marijuana is at the forefront of global controversy debating the appropriateness of its extensive illegal status. In most Union states it's grown legalized for medical purposes. This pattern is known as "medical marijuana" and is highly applauded by advocates while simultaneously loathed harshly by opponents (Dubner, 2007; Nakay, 2007; Van Tuyl, 2007). It is in this particular context which it had been decided to decide on the subject of the pharmacological and physical effects of marijuana for the basis of the research article.
What is marijuana?
Marijuana is a plant more correctly known as cannabis sativa. As mentioned, several cannabis sativa plants do not have abuse potential and are defined as hemp. Eagle Hemp CBD Gummies ( Recommended Browsing - https://www.bainbridgereview.com/national-marketplace/eagle-hemp-cbd-gum... ) is used widely for various fiber products including newspaper and artist's canvas. Cannabis sativa with abuse potential is what we call marijuana (Doweiko, 2009). It's interesting to note that though widely studies for many years, there's a whole lot that researchers still do not understand about marijuana. Neuroscientists and biologists - http://www.recruitingblogs.com/main/search/search?q=biologists know what the consequences of marijuana are however, they still don't completely understand exactly why (Hazelden, 2005).
Deweiko (2009), Gold, Frost Pineda, & Jacobs (2004) point out that of roughly four hundred acknowledged chemicals found in the cannabis plants, scientists know of over sixty which are thought to have psychoactive effects on the human brain. The most recognized also powerful of these is â-9-tetrahydrocannabinol, or THC. Like Hazelden (2005), Deweiko claims that while we know most of the neurophysical effects of THC, the reasons THC produces these consequences are unclear.
Neurobiology:
As a psychoactive substance, THC specifically influences the main nervous system (CNS). It affects an immense variety of neurotransmitters and catalyzes other biochemical as well as enzymatic exercise as well. The CNS is stimulated when the THC activates certain neuroreceptors in the brain causing the various physical and emotional reactions that will be expounded on more specifically further on. The sole substances that can activate neurotransmitters are substances which mimic chemicals the brain produces by natural means. The fact that THC stimulates brain function teaches scientists that the brain has natural cannabinoid receptors. It's still unclear the reason why humans have organic cannabinoid receptors as well as the way they work (Hazelden, 2005; Martin, 2004). What we do know is the fact that marijuana will stimulate cannabinoid receptors around 20 times more actively than the body's natural neurotransmitters ever could (Doweiko, 2009).
Perhaps the biggest mystery on the planet is the connection between THC and the neurotransmitter serotonin. Serotonin receptors are of all the most stimulated by just about all psychoactive drugs, but most especially alcohol and nicotine. Impartial of marijuana's connection with the substance, serotonin is already somewhat understood neurochemical and its supposed neuroscientific roles of functioning and purpose continue to be mostly hypothetical (Schuckit & Tapert, 2004). What neuroscientists have found definitively is that marijuana smokers have really high levels of serotonin activity (Hazelden, 2005). I would hypothesize it may be this connection between THC and serotonin that explains the "marijuana maintenance program" of achieving abstinence from alcoholic drinks and makes it possible for marijuana smokers to stay away from painful withdrawal symptoms and also stay away from cravings from alcohol. The efficacy of "marijuana maintenance" for helping alcohol abstinence isn't scientific but is a phenomenon I've really experienced with quite a few clients.
Interestingly, marijuana mimics so many neurological reactions of other drugs that it is incredibly difficult to classify in a specific class. Researchers will place it in virtually any of these categories: psychedelic; hallucinogen; or maybe serotonin inhibitor. It has qualities which mimic similar chemical responses as opioids. Other chemical reactions mimic stimulants (Ashton, 2001; Gold, Frost-Pineda, & Jacobs, 2004). Hazelden (2005) classifies marijuana in its own special class - cannabinoids. The reason for this confusion is the sophistication of the numerous psychoactive qualities found within marijuana, both known and unknown. One recent customer I saw could not recover from the obvious distortions he suffered as an outcome of pervasive psychedelic use so long as he was nonetheless smoking marijuana. This appeared to be as a consequence of the psychedelic properties located within energetic cannabis (Ashton, 2001). Though not durable adequate to produce these visible distortions alone, marijuana was sturdy enough to keep the brain from healing and recovering.
Emotions:
Cannibinoid receptors are placed throughout the brain thus affecting a multitude of functioning. The most important on the mental level is the stimulation of the brain's nucleus accumbens perverting the brain's natural reward centers. An alternative is the fact that of the amygdala which regulates one's emotions as well as fears (Adolphs, Trane, Damasio, & Damaslio, 1995; Van Tuyl, 2007).